Light chain cardiac amyloidosis in a nonagenarian
Koji Takahashi1
Mina Yamashita2
Tomoki Sakaue1
Daijiro Enomoto2
Shigeki Uemura2
Takafumi Okura2
Shuntaro Ikeda1
Takanori Senba3
Akira Saijo4
Nobuhisa Yamamura4
Sohei Kitazawa5
1.Department of Community Emergency Medicine,Ehime University Graduate School of Medicine,Ehime,Japan;Department of Cardiology,Yawatahama City General Hospital,Ehime,Japan2.Department of Cardiology,Yawatahama City General Hospital,Ehime,Japan3.Department of Hematology,Yawatahama City General Hospital,Ehime,Japan4.Department of Clinical Pathology,Yawatahama City General Hospital,Ehime,Japan5.Department of Molecular Pathology,Ehime University Graduate School of Medicine,Ehime,Japan
摘要：Cardiac amyloidosis is an infiltrative and restrictive cardiomyopathy caused by the extracellular deposition of amyloid fibrils within the heart as systemic amyloidosis, leading to heart failure, reduced quality of life, and death.[1]There are two major amyloid fibril proteins that affect the heart: amyloid immunoglobulin light chain (AL) and amyloid transthyretin (ATTR). The latter is further subdivided into wild-type ATTR and variant types based on the presence of a mutation in the transthyretin gene.
机标关键词：lightchainamyloidosiscardiacnonagenarian
论文发表日期：2022-01-28
在线出版日期：2025-08-15（本平台首次上网日期，不代表文献的发表时间）
页数：7( 83-89 )