颈性眩晕发病机制新进展
张阳1
李放2
孙天胜3
1.100853 北京,解放军医学院;100700 北京,陆军总医院骨科2.陆军总医院骨科, 北京,1007003.解放军医学院, 北京,100853
摘要：The pathogenesis of cervical vertigo is still not clear. Currently there are several hypotheses including proprioceptive cervical vertigo, vertebral artery compressive hypothesis, neurohumor factor hypothesis,cervical sympathetic nerve related vertigo and Migraine associated cervicogenic vertigo. These hypotheses tried to explain the pathogenesis of cervical vertigo from different perspectives, however, they all failed to reveal it completely. Neck proprioceptive cervical vertigo hypothesis indicated that abnormal afferent input to the vestibular nucleus from damaged joint receptors in the upper cervical region could alter the vestibular nuclei of the brainstem, resulting in cervical vertigo. Vertebral artery compressive hypothesis thought that compression of the vertebral artery might lead to vertebrobasilar insufficiency, initiate vestibular organ ischemia, thus result in vertigo. Some authors indicated that neurohumor factors played an important role in the pathogenesis of cervical vertigo. Cervical sympathetic nerve related vertigo hypothesis suggested that sympathetic plexus surrounding the vertebral arteries could be stimulated by cervical degenerative disease. And this stimulation could contribute to the reflexive vasoconstriction of the vertebrobasilar system, thus accounting for the cervical vertigo. Migraine associated cervicogenic vertigo hypothesis postulated that there were relations between migraine and cervicogenic vertigo. The reciprocal connections between the vestibular nuclei and the trigeminal nucleus caudalis can provide a mechanism in which the vestibular signals would influence the vascular trigeminal paths, widely related to the processing of the vestibular and trigeminal information during the migraine attacks.
关键词：眩晕本体感觉椎基底动脉供血不足体液偏头痛综述
分类号：R441.2(诊断学)R681.5(骨科学（运动系疾病、矫形外科学）)
论文发表日期：2018-01-01
在线出版日期：2025-08-15（本平台首次上网日期，不代表文献的发表时间）
页数：4( 373-376 )
英文信息