Heat shock protein 90 is a potential therapeutic target for ameliorating skeletal muscle abnormalities in Parkinson’s disease
Nour Erekat
Ahed Al-Khatib
Muhammed Al-Jarrah
摘要:Previous studies have conifrmed that heat shock protein 90 overexpression can lead to dopami-nergic neuronal death. This study was designed to further investigate what effects are produced by heat shock protein 90 after endurance exercise training. Immunohistochemistry results showed that exercise training signiifcantly inhibited heat shock protein 90 overexpression in the soleus and gastrocnemius in Parkinson’s disease rats, which is a potential therapeutic target for ameliorating skeletal muscle abnormalities in Parkinson’s disease.
机标关键词:heat shock proteinendurance exerciseskeletal muscleneuronal death
论文发表日期:2014-01-01
在线出版日期:2025-08-15(本平台首次上网日期,不代表文献的发表时间)
页数:6( 616-621 )
英文信息展开
中国神经再生研究(英文版)

中国神经再生研究(英文版)

CSTPCDSCI
ISSN:1673-5374
年,卷(期):2014,(6)
所属栏目:RESEARCH AND REPORTS