Time dependent integration of matrix metalloproteinases and their targeted substrates directs axonal sprouting and synaptogenesis following central nervous system injury362-376
In the presence of danger:the extracellular matrix defensive response to central nervous system injury377-384
Characterization of hippocampal Cajal-Retzius cells during development in a mouse model of Alzheimer’s disease (Tg2576)394-401
The Pael-R gene does not mediate the changes in rotenone-induced Parkinson’s disease model cells402-406
